ETIOLOGY OF HYPOVITAMINOSES A AND D IN YOUNG ANIMALS

Аннотация

This in the article Age animals A and D hypovitaminosisI with sick animals growth and from development stay , lose weight, slime floors pale, skin dry, wool cover glitter loss, bones mortification with passing substance exchange etiology according to information cited . Key words: hypovitaminosis A, hypovitaminosis D, carotene, Rhodopsin, Rickets, deformity, calcium and phosphorus. Login. Uzbekistan In the Republic livestock field and his/her networks develop for 2022-2026 program confirmation about in the decision : Today on the day in our republic total 18,032 livestock farms there is of which 7,614 cattle breeding, 3,263 sheep and goat farming , 142 horse breeding , 52 camel farming , 1,163 poultry farming , 4,829 fishing , 715 beekeeping and 254 rabbit farming in the direction of organization done farms support support measure events by designating placed. Subject relevance. Today on the day in our republic total 7,614 cattle breeding if their some growth and from development to remain, to lose weight , to slim down floors pale , skin dry, wool cover glitter loss of bones mortification such as situations prevent to take current task is considered . Literature analysis: Hypovitaminosis and Avitaminosis A. (A hypovitaminosis ) Animals everyone avitaminosis inside the most many scattered , it is in the calf, the pig in children, in the hands and in chicks often, in mares and less Vitamin A is produced in the dark . to see providing rhodopsin biosynthesis for necessary . Rhodopsin eye net on the curtain of sticks light sensor pigment is considered. Light fall as a result pigment broken down , nerve impulses harvest to be provides. Reasons. Vitamin A in the diet or his/her provitamin calculated carotene amount animal need Endogenous hypovitaminosis A hepatitis, liver cirrhosis, gastroenteritis, some contagious and invasion diseases or chronic poisonings during observation possible Etiology: Vitamin A deficiency active shape calculated retinol eye ( retina) opsin protein in the membrane with from the merger rhodopsin ( see purple ( h ) will be and that eye into the darkness adapt provides. When vitamin A is deficient skin , eye young glands , conjunctiva , respiratory paths , digestion channel and urogenital members in the epithelium hyperplasia and branching develops , as well as mucus curtains h gesture functions decrease , ovary and of the testicular epithelium metaplasia and to dystrophy follicles atrophy , spermiogenesis fading , skin epithelium hyperplasia and branching , sweat and sebaceous glands atrophy develops . Sexual members epithelium injury of the murtak to die and baby to throw reason to be possible . Eye horn of the curtain drying up to remain and of sight decrease specific sign is considered . 101 ILM-FAN VA INNOVATSIYA ILMIY-AMALIY KONFERENSIYASI in-academy.uz/index.php/si Hypovitaminosis D is a disease of young growing animals associated with a deficiency of vitamin D, an imbalance of phosphorus and calcium in the body. It leads to the development of rickets. Rickets can also occur against the background of a deficiency in the body of other vitamins, vital micro- and macroelements, as well as with ultraviolet radiation and gastrointestinal diseases. Causes. The main causes of rickets in young cattle are: vitamin D deficiency, imbalance or deficiency of calcium and phosphorus in the body of the young animal, and keeping them in damp and cold rooms. Etiology. Avitaminosis D, or rickets, occurs as a result of insufficient vitamin D in the diet and prolonged exposure of animals to ultraviolet rays. Rickets is also caused by an imbalance in the ratio of calcium and phosphorus salts and their insufficient amount in the diet. Figure 1. Skin pathology in an animal with hypovitaminosis A Research object and methods: The study was conducted on young calves of the “Jamol Ota” cattle breeding farm, Yangiyul city, Tashkent region. The "Jamol Ota" farm has a total area of 5 hectares and has 230 head of cattle, of which 130 are Holstein cows for dairy farming and 35 are 6-month-old calves. When examining the area allocated for calves, it was found that there was poor sunlight and a malfunction in the air circulation system. When examining their feed, they were only given up to 3 liters of milk twice a day, and bran was given as a supplement. During clinical examination of calves, signs such as unsatisfactory general condition of calves, growth and development delay, dullness of wool cover, decreased elasticity of wool, loss of appetite, calves licking each other, the floor and various objects are observed. Calves have difficulty walking, pain in the joints and minor mechanical injuries can also lead to fractures of the tubular bones. The calf's spine and tubular bones are curved, and the calves are uncomfortable due to pain when palpated. In order to diagnose the disease in a comprehensive manner, blood samples were taken from 5 calves up to 6 months of age, based on anamnesis data, recording of disease-