К содержанию
РецензированоEN2025

ORIGIN AND MODERN CLINICAL DIAGNOSIS OF INFILTRATIVE HEART DISEASES

Davlat Ergashev · Mehroj Xolmatov · Husan Boltayev · Akmal Mardonqulov · Asomiddin Yavg'ashev

Современная наука и исследования · 199-203 · 2025

Аннотация

To date, nine amyloidogenic proteins have been identified that can accumulate in the myocardium and cause cardiac amyloidosis. Three of them are common: serum amyloid A (AA amyloidosis, a consequence of chronic inflammation and infectious diseases), immunoglobulin light chains (AL amyloidosis, a consequence of monoclonal gammopathy), and transthyretin (two variants of transthyretin - ATTR amyloidosis: ATTRv amyloidosis, here ATTRv amyloidosis is associated with a non-hereditary type of transthyretin amyloidosis).

Доказательный фрагмент

«To date, nine amyloidogenic proteins have been identified that can accumulate in the myocardium and cause cardiac amyloidosis.»

Образец извлечения

Демо-превью

To date, nine amyloidogenic proteins have been identified that can accumulate in the myocardium and cause cardiac amyloidosis. Полный текст публикации CiteX не хранит — доступ к первоисточнику открывается по DOI согласно лицензии издателя и выбранному плану.

DOI не указан
Цитирований
0
Тип
Журнал
Страна
Узбекистан
Ключевые слова
The prognosis of cardiac amyloidosis is very unfavorable. However, in recent years, certain progress has been made not only in diagnostics, but also in the development of drug therapy for cardiac amyloidosis, so that experts from the Working Group on Myocardial and Pericardial Diseases of the European Society of Cardiology have developed a consensus document on the diagnosis and treatment of cardiac amyloidosis [1].
Спросить CiteX об этой работе

Смежные источники