Острая эзофагогастродуоденопатия при травматической болезни. Морфогенез и частная патология эрозивных повреждений

Аннотация

In the study of biopsies obtained during fibrogastroduodenoscopy (43 men, 268 biopsies) and autopsy (9 people) affected by severe concomitant injury that centralization of blood circulation at the level of the digestive system that leads to local circulatory hypoxia with morphological changes ranging from mild reversible ischemia to necrosis were detected. Polietiology and monopathogenesis of shock in general and traumatic disease in particular, mean flexibility of mechanisms of alterative components development. Morphologically stressful, immunopathological and circulatory changes are expressed in the development of acute esophagogastroduodenopathy in form of acute erosions and ulcers with severe hemorrhagic component. Clinically it is manifested in the form of dynamic ileus, dyskinesia of the stomach and duodenum, pyloric insufficiency of duodenal bile reflux